We proliferate after birth—the length rises by around 50% during infancy, and the weight doubles. Then, development slows and enters a continuous period in childhood until a growth spike in adolescence. But why is this so?
Researchers discovered the solution. They look at the DNA of 30,000 children and their parents from the Norwegian mother, father, and child cohort. Millions of genetic polymorphisms from each individual were evaluated and connected to growth data from height and weight measures taken from birth to eight years of age.
The study’s results were published in the periodical ‘Nature Metabolism.’ It has offered insight into the systems that govern hunger and energy metabolism early in life, which may aid in the development of improved treatments for obesity in adolescence and maturity.
“It was discovered that genes connected to excessive obesity, hunger, and the body’s energy consumption are in charge of growth control.”
“This is dynamic in the sense that individual genes influence only on certain stages of growth.” “We recognize that the reasons why parents have always noticed that some infants are born with a naturally stronger appetite than others and have much more fat mass in infancy,” he added.
“These dynamic effects are particularly relevant in the initial years of life and do not raise the risk of subsequent obesity,” Njolstad noted.
Some of the genes have been connected to medications that are being tried to reduce weight growth in people who are extremely obese. As a result, the findings may have implications for the management of normal obesity.
The authored article is written by Sejal Wakkar and shared with Prittle Prattle News exclusively.
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